ATG5
autophagy related 5
- Ensembl:
- ENSG00000057663
- UniProt:
- Q9H1Y0
- OMIM:
- 604261
- Synonyms:
- APG5, APG5L, ASP, HAPG5
Cilia effects upon perturbation of ATG5
- Cilia number / % ciliated:
- Decreased cilia number
- Loss-of-function effect:
- Shorter cilia
Ciliogenesis screen results (4 screens)
- Kim2016: Not Reported
- Wheway et al. 2015 (siRNA) [siRNA]: Ciliogenesis Defect (z=-5.32) PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
- Roosing et al. 2015 (siRNA) [siRNA]: No effect PMID:26595381
Subcellular localization
basal body, cilia
Functional category
- Ciliary assembly/disassembly
- Actin & cytoskeleton regulation
- T cell biology
- Viral interactions
- Protein processing & maturation
- Signaling (Hedgehog, GPCRs, ion channels)
- Cardiac & muscle development
- Muscle contraction & physiology
Function
OFD1 at the centriolar satellites is rapidly degraded by autophagy upon serum starvation. In autophagy- deficient Atg5 or Atg3 null mouse embryonic fibroblasts, OFD1 accumulates at centriolar satellites, leading to fewer and shorter primary cilia and a defective recruitment of BBS4 (Bardet-Biedl syndrome 4) to cilia. MUtations cause spinocerebellar ataxia 25.The percentage of Atg5−/− cells that form a primary cilium, as compared to Atg5+/+ cells, was significantly reduced and the cilia that did form were shorter (PMID: 24089205).