CEP295
centrosomal protein 295
- Ensembl:
- ENSG00000166004
- UniProt:
- Q9C0D2
- OMIM:
- 617728
- Synonyms:
- KIAA1731
Cilia effects upon perturbation of CEP295
Ciliogenesis screen results (1 screen)
- Breslow et al. 2018 (CRISPR) [CRISPR]: Positive Regulator (Hh signaling, casTLE effect=-1.44) PMID:29459680
Phenotypes
- Mouse phenotype:
- abnormal embryo size, preweaning lethality, incomplete penetrance, decreased bone mineral content
- Mouse ciliopathy phenotype:
- male infertility
Ciliopathy associations
- Seckel-like Syndrome
Subcellular localization
basal body, centrosome
Functional category
- Ciliary assembly/disassembly
- Actin & cytoskeleton regulation
Function
Depletion of CEP295 resulted in a decrease in the numbers of centrioles and centrosomes and triggered p53-dependent G1 cell cycle arrest, and the loss of CEP295 causes extensive primary ciliary defects in both patient-derived fibroblasts and RPE1 cells(PMID: 38154379).
Model organism evidence
Drosophila (1 reference)
We conclude that Ana1 helps recruit Polo to mother centrioles to specifically promote mitotic centrosome assembly and centriole elongation in G2, but not centriole duplication, centriole disengagement or cilia assembly.
PMIDs: 34156068