GNAI1
G protein subunit alpha i1
- Ensembl:
- ENSG00000127955
- UniProt:
- P63096
- OMIM:
- 139310
- Synonyms:
- GNAI1
Cilia effects upon perturbation of GNAI1
- Cilia number / % ciliated:
- Decreased cilia number
- Loss-of-function effect:
- Impaired ciliogenesis
Ciliogenesis screen results (4 screens)
- Kim2016: Not Reported
- Wheway et al. 2015 (siRNA) [siRNA]: No effect PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
- Roosing et al. 2015 (siRNA) [siRNA]: No effect PMID:26595381
Phenotypes
- Mouse phenotype:
- abnormal spleen morphology, hypospermia
- Human ciliopathy phenotype:
- Mutations cause developmental delay, intellectual disability, hypotonia, epilepsy.
Ciliopathy associations
- Intellectual Developmental Disorder
Subcellular localization
basal body, centrosome, nucleus
Functional category
- Ciliary assembly/disassembly
- Trafficking (BBSome, small GTPases, vesicular transport, ATPases)
- Actin & cytoskeleton regulation
- Metabolism
- Viral interactions
- Cell migration & adhesion
- Signaling (Hedgehog, GPCRs, ion channels)
Function
G I1 has localized in basal body. GNAI1 knockdown impairs ciliogenesis in human cells. GNAI1 variants disrupt ciliary localization of Gαi1 proteins. (40894620)
Model organism evidence
C. elegans (2 references)
Here, we show that GNAI1 is required for ciliogenesis in human cells and use Caenorhabditis elegans as a whole-organism model to determine the functional impact of 7 GNAI1-disorder patient variants.
Here, we show that GNAI1 is required for ciliogenesis in human cells and use C.