KIF9
kinesin family member 9
- Ensembl:
- ENSG00000088727
- UniProt:
- Q9HAQ2
- OMIM:
- 607910
- Synonyms:
- MGC104186
Cilia effects upon perturbation of KIF9
- Cilia number / % ciliated:
- No effect
- Loss-of-function effect:
- Shorter cilia
- Overexpression effect:
- Unknown
Ciliogenesis screen results (3 screens)
- Wheway et al. 2015 (siRNA) [siRNA]: No effect PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
- Roosing et al. 2015 (siRNA) [siRNA]: No effect PMID:26595381
Phenotypes
- Mouse phenotype:
- small spleen, small liver, developmental dysplasia, abnormal skin morphology
- Mouse ciliopathy phenotype:
- abnormal liver morphology, male infertility
Subcellular localization
basal body, cilia
Functional category
- Ciliary assembly/disassembly
- Actin & cytoskeleton regulation
- Metabolism
- T cell biology
- Reproduction & sperm
- Cell migration & adhesion
Function
Kif9 is necessary for ciliary motility and the proper distal localization of not only central pair proteins, but also radial spokes and dynein arms (35531639). Kif9 is integral for ciliary beating and is necessary for proper axonemal distal end integrity.(PMID: 35531639) KIF9 loss causes centriolar satellite aggregation near centrosome, leading to cilia length defects. Alters levels of TALPID3, CEP131, CEP170, and CEP290. Disrupts cilia length without necessarily affecting ciliation frequency.
Model organism evidence
The highly conserved kinesin family member 9 (KIF9) localizes to the central microtubule pair in the flagella of Chlamydomonas cells.
Kif9 is an active kinesin motor required for ciliary beating and proximodistal patterning of motile axonemes.
Kif9 is an active kinesin motor required for ciliary beating and proximodistal patterning of motile axonemes.
PMIDs: 35531639