LRRC56
leucine rich repeat containing 56
- Ensembl:
- ENSG00000273831, ENSG00000161328
- UniProt:
- Q8IYG6
- OMIM:
- 618227
- Synonyms:
- DKFZP761L1518, DNAAF12, FLJ00101, ODA8
Cilia effects upon perturbation of LRRC56
- Cilia number / % ciliated:
- No effect
- Loss-of-function effect:
- No effect
Ciliogenesis screen results (3 screens)
- Wheway et al. 2015 (siRNA) [siRNA]: No effect PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
- Pusapati et al. 2018 (CRISPR) [CRISPR]: Negative Regulator (Hh signaling, pos_rank=426, lfc=-0.12) PMID:30270045
Phenotypes
- Mouse phenotype:
- hypospermia, abnormal heart position or orientation, abnormal heartbeat, spermatogenesis defect, germ cell defect, enlarged lymph nodes, abnormal skin morphology, developmental dysplasia, abnormal lung morphology
- Mouse ciliopathy phenotype:
- increased circulating alanine transamise level, male infertility, hydrocephalus
Ciliopathy associations
- Hydrocephalus
- Male Infertility
- Mucociliary Clearance Disorder
- Primary Ciliary Dyskinesia
- Visceral Heterotaxy / Situs Inversus
Subcellular localization
basal body, cilia, lysosomes
Functional category
- Ciliary assembly/disassembly
- Cell migration & adhesion
Function
Interacts with the intraflagellar transport (IFT) protein IFT88. Required for the transport and addition of dynein arms to the distal end of the flagellum during cilia assembly. Mutations result in a disease entity within the group of mucociliary clearance disorders and loss of ciliary motility (30388400). LRRC56 frameshift variant causes severely dyskinetic cilia without obvious ultrastructural defects. Lrrc56-KO mice show laterality defects, male infertility, hydrocephalus, and defective mucociliary clearance. Reduces expression of MIPs and axonemal dynein assembly factors.
Model organism evidence
LRRC56 deficiency cause motile ciliopathies in humans and mice.
LRRC56 deletion causes primary ciliary dyskinesia in mice characterized by dynein arms defects.
Physical and functional interaction of the ciliopathy proteins Lrrc56 and Odad3 control deployment of axonemal dyneins in vertebrate multiciliated cells.
Physical and functional interaction of Lrrc56 and Odad3 controls deployment of axonemal dyneins in vertebrate multiciliated cells.