MAPKBP1

mitogen-activated protein kinase binding protein 1

Ensembl:
ENSG00000137802
UniProt:
O60336
OMIM:
616786
Synonyms:
KIAA0596, NPHP20

Cilia effects upon perturbation of MAPKBP1

Cilia number / % ciliated:
Decrease
Loss-of-function effect:
Shorter cilia

Ciliogenesis screen results (2 screens)

  • Wheway et al. 2015 (siRNA) [siRNA]: Ciliogenesis Defect (z=-2.42) PMID:26167766
  • Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680

Phenotypes

Mouse phenotype:
increased blood urea nitrogen level, impaired glucose tolerance, increased circulating phosphate level, increased spleen weight
Mouse ciliopathy phenotype:
increased heart weight, increased kidney weight
Human ciliopathy phenotype:
nephronophthisis; nephronophthisis 20

Ciliopathy associations

  • Nephronophthisis

Subcellular localization

basal body

Functional category

  • Ciliary assembly/disassembly
  • Actin & cytoskeleton regulation

Function

While wild-type MAPKBP1 exhibited centrosomal, basal body, and microtubule association, mutant proteins lost the latter and showed reduced recruitment to cell cycle dependent centriolar structures (32505465). MAPKBP1/NPHP20 is lost from ciliary basal bodies in NPHP20 patients; knockdown causes shortened primary cilia. JNK activation leads to MAPKBP1 dissociation from basal body and cilia disassembly. Actin pharmacological restoration rescues ciliary length.

Model organism evidence

Mus musculus (2 references)

Ocular manifestations of renal ciliopathies.

The association of microcephaly protein WDR62 with CPAP/IFT88 is required for cilia formation and neocortical development.

PMIDs: 37644229, 31816041