SMAD2
SMAD family member 2
- Ensembl:
- ENSG00000175387
- UniProt:
- Q15796
- OMIM:
- 601366
- Synonyms:
- JV18-1, MADH2, MADR2
Cilia effects upon perturbation of SMAD2
- Cilia number / % ciliated:
- No effect
- Loss-of-function effect:
- No effect
Ciliogenesis screen results (4 screens)
- Kim2016: Not Reported
- Wheway et al. 2015 (siRNA) [siRNA]: Ciliogenesis Defect (z=-11.71) PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
- Roosing et al. 2015 (siRNA) [siRNA]: No effect PMID:26595381
Phenotypes
- Human ciliopathy phenotype:
- congenital heart defects, multiple types, 8, with or without heterotaxy
Subcellular localization
basal body, cytosol, nucleus, transition zone
Functional category
- Ciliary assembly/disassembly
- Signaling (Hedgehog, GPCRs, ion channels)
- Transition zone
- Transcription regulation
- Cilia length regulation
Function
Madiator in the activation of TGF-尾 sig ling pathway and controls cilia length (25959824). Downstream of TGFBR1 and 2, which phosphorylate SMAD2/3 at base of cilium upon TGF-尾 stimulation (23746451).
Model organism evidence
Danio rerio (1 reference)
Additionally, we found that Kupffer's vesicle formation and ciliogenesis were impaired following gdf11 deletion.
PMIDs: 39407407
Mus musculus (1 reference)
Primary cilia abnormalities participate in the occurrence of spontaneous abortion through TGF-β/SMAD2/3 signaling pathway.
PMIDs: 38704705