WNT11
Wnt family member 11
- Ensembl:
- ENSG00000085741
- UniProt:
- O96014
- OMIM:
- 603699
Cilia effects upon perturbation of WNT11
Ciliogenesis screen results (2 screens)
- Wheway et al. 2015 (siRNA) [siRNA]: No effect PMID:26167766
- Breslow et al. 2018 (CRISPR) [CRISPR]: No Significant Effect PMID:29459680
Phenotypes
- Mouse phenotype:
- decreased body length, increased blood urea nitrogen level
- Mouse ciliopathy phenotype:
- small testis, abnormal kidney morphology, abnormal testis morphology
Subcellular localization
cilia associated gene
Functional category
- Ciliary assembly/disassembly
- Signaling (Hedgehog, GPCRs, ion channels)
- Cardiac & muscle development
- ECM & connective tissue
- Transcription regulation
Function
Nlz1 contributes to motile cilia formation by positively regulating Wnt11/presumed non-canonical Wnt sig ling ( 26327644).
Model organism evidence
Primary cilia are located at the dendritic tips of sensory neurons and house the molecular machinery necessary for detection and transduction of sensory stimuli.
PMIDs: 33460640
Furthermore, Nlz1 contributes to motile cilia formation by positively regulating Wnt11/presumed non-canonical Wnt signaling.
Laterality was randomized in the brain, heart and viscera, likely a consequence of shorter cilia with disrupted axonemes and perturbed intravesicular fluid flow in Kupffer's vesicle.
Remarkably, in addition to cyst formation, loss of primary cilia in the ureteric epithelial cell leads to decreased expression of Wnt11 and Ret and reduced ureteric branching.
This was preceded by excessive cell proliferation and apoptosis, and accompanied by disrupted ciliogenesis within the ureteric bud epithelium.